Preliminary scientific studies point out conolidine may well inhibit certain ion channels, cutting down neuronal excitability and restricting suffering signals. This mechanism is particularly suitable in neuropathic agony, where by abnormal signaling causes persistent pain. Additionally, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to suffering percep... https://pikd382fed8.wikicorrespondent.com/user